What is the difference between svo2 and scvo2

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In case of sale of your personal information, you may opt out by using the link Do not sell my personal information. Cookies Policy. It is basically the percentage of oxygen remaining in the venous blood returning to the right side of the heart. This is the oxygen left over in the blood after supplying all the parts of the body except the head. Scvo2 stands for central venous oxygen saturation.

It is the oxygen saturation of venous blood coming from the head and upper body. It is measured from the superior vena cava, that drains blood from the head and upper body to the heart and thus, it is called as central venous oxygen saturation.

This is because the lower half of the body extracts less oxygen and the brain extracts more oxygen than other organs of the body. Together, both the saturation percentages give us knowledge about the balance between the delivery of oxygen and oxygen consumption in the body.

The procedure for assessing Scvo2 is less risky and has far lesser complications than measuring Svo2. While collecting blood samples for checking Scvo2, the blood is collected from the superior vena cava by placing a thin, fibreoptic central venous catheter in the jugular vein. In case of Svo2, the value is assessed by taking an average by collecting 3 samples from three different regions- first sample from the lower limbs, second sample combined from head and upper limbs and third from cardiac venous supply.

If not an average, blood sample can be directly taken from the pulmonary artery. A pulmonary artery catheter is used for this procedure. The pulmonary artery carries the venous blood from the right ventricle of the heart to the lungs for oxygenation.

Taking sample from this artery is a highly invasive procedure and hence, has more chances of complications. This is a major difference between collection of sampled for Svo2 and Scvo2. Shock results from serious illness compromising either vascular muscle tone most commonly septic shock , the heart's function, or the volume of plasma inside blood vessels. The true goal of treatment for shock is to correct the underlying cause, but except for some causes of shock STEMI, hypovolemia that's not usually immediately possible.

Interim goals for treatment for shock are to augment perfusion and oxygen delivery and minimize organ damage until the body's natural homeostatic mechanisms return. While simple in theory, the complexity of the body's response to shock and its therapies can make realtime goal-directed management of shock surprisingly challenging.

Restoring systemic blood pressure to a mean arterial pressure between 65 - 70 mm Hg is "a good initial goal," but this threshold pressure should not be considered an absolute. Some patients with uncontrolled hypertension at baseline may require a higher MAP goal, for example. On the other hand, healthy people with normotension at baseline and pure hypovolemia from a GI bleed may tolerate MAPs lower than 65 mm Hg while awaiting rapid blood transfusion.

Once an acceptable MAP value is achieved with fluid resuscitation and vasopressors, proper management of shock necessitates continued close attention to mental status, urine output, and skin appearance and temperature, along with laboratory values. Urine output particularly should be followed closely with such interventions goal 0. Although cardiac output is a major driver for oxygen delivery to tissues, "the optimal cardiac output is difficult to define.

Further, the "optimal" cardiac output can change in the same patient over time, and can vary widely between patients.